By MD Frank H. Netter
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2007). , 2006; Straub and Weinberger, 2006). Several of these target the NMDA receptor and control proteins that act to strengthen the synapse. Dysfunction of NMDA control proteins can result in hypofunction of the NMDA synapse: the NMDA receptor hypofunction hypothesis of schizophrenia (Stahl, 2007a). Weak synapses with AMPA receptors may be pruned, but this process takes time. In addition, synaptic pruning is very active during adolescence. This may explain why schizophrenia onset is associated with the adolescent period of life (Stahl, 2007b).